Expressions of endocan in patients with meningiomas and gliomas
dc.contributor.author | Atukeren, Pınar | |
dc.contributor.author | Kunbaz, Ahmad | |
dc.contributor.author | Türk, Okan | |
dc.contributor.author | Kemerdere, Rahsan | |
dc.contributor.author | Ulu, Mustafa Onur | |
dc.contributor.author | Tanrıverdi, Taner | |
dc.contributor.buuauthor | İnanır, Nursel Türkmen | |
dc.contributor.department | Uludağ Üniversitesi/Tıp Fakültesi/Adli Tıp Anabilim Dalı. | tr_TR |
dc.contributor.orcid | 0000-0002-4047-6455 | tr_TR |
dc.contributor.scopusid | 57188706721 | tr_TR |
dc.date.accessioned | 2022-12-13T12:52:28Z | |
dc.date.available | 2022-12-13T12:52:28Z | |
dc.date.issued | 2016-06-30 | |
dc.description.abstract | Objective. Endocan has been shown to be a marker for several cancers and may show degree of malignancy. The aim of this study is to assess tissue levels of endocan in common brain tumors, namely, meningiomas, low-grade gliomas (LGGs), and high-grade gliomas (HGGs). Patients and Methods. Endocan was assayed by commercially available enzyme linked immunosorbent assay (ELISA) kits in a total of 50 brain tumors (20 meningiomas, 19 LGGs, and 20 HGGs) and 15 controls. The results were compared to control brain tissues. Results. Each tumor group showed significant higher levels of endocan compared to controls (p < 0.05). In addition, endocan levels showed steady increase from the least (meningiomas) to the most (HGGs) malignant tumors and positive correlation was noted between the degree of malignancy and endocan level (p = 0.0001). Conclusion. Endocan, a vital molecule for angiogenesis, is expressed in common brain tumors and results suggest that endocan could be a marker for malignancy. | en_US |
dc.description.sponsorship | İstanbul Üniversitesi - 55673 | tr_TR |
dc.identifier.citation | Atukeren, P. vd. (2016). "Expressions of endocan in patients with meningiomas and gliomas". ed. Lance A. L. Disease Markers, 2016. | en_US |
dc.identifier.issn | 0278-0240 | |
dc.identifier.issn | 1875-8630 | |
dc.identifier.pubmed | 27528791 | tr_TR |
dc.identifier.scopus | 2-s2.0-84982153572 | tr_TR |
dc.identifier.uri | https://doi.org/10.1155/2016/7157039 | |
dc.identifier.uri | https://www.hindawi.com/journals/dm/2016/7157039/ | |
dc.identifier.uri | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4978841/ | |
dc.identifier.uri | http://hdl.handle.net/11452/29860 | |
dc.identifier.volume | 2016 | tr_TR |
dc.identifier.wos | 000381130500001 | tr_TR |
dc.indexed.pubmed | PubMed | en_US |
dc.indexed.scopus | Scopus | en_US |
dc.indexed.wos | SCIE | en_US |
dc.language.iso | en | en_US |
dc.publisher | Hindawi | en_US |
dc.relation.collaboration | Yurt içi | tr_TR |
dc.relation.collaboration | Sanayi | tr_TR |
dc.relation.journal | Disease Markers | en_US |
dc.relation.publicationcategory | Makale - Uluslararası Hakemli Dergi | tr_TR |
dc.rights | info:eu-repo/semantics/openAccess | en_US |
dc.subject | Biotechnology & applied microbiology | en_US |
dc.subject | Genetics & heredity | en_US |
dc.subject | Research & experimental medicine | en_US |
dc.subject | Pathology | en_US |
dc.subject | Cell-specific molecule-1 | en_US |
dc.subject | Marker | en_US |
dc.subject | Esm-1 | en_US |
dc.subject | Survival | en_US |
dc.subject | Invasion | en_US |
dc.subject | Cancer | en_US |
dc.subject.emtree | Anticonvulsive agent | en_US |
dc.subject.emtree | Endocan | en_US |
dc.subject.emtree | Protein | en_US |
dc.subject.emtree | Unclassified drug | en_US |
dc.subject.emtree | ESM1 protein, human | en_US |
dc.subject.emtree | Proteoglycan | en_US |
dc.subject.emtree | Tumor marker | en_US |
dc.subject.emtree | Tumor protein | en_US |
dc.subject.emtree | Adult | en_US |
dc.subject.emtree | Article | en_US |
dc.subject.emtree | Cancer chemotherapy | en_US |
dc.subject.emtree | Cancer radiotherapy | en_US |
dc.subject.emtree | Clinical article | en_US |
dc.subject.emtree | Comparative study | en_US |
dc.subject.emtree | Controlled study | en_US |
dc.subject.emtree | Enzyme linked immunosorbent assay | en_US |
dc.subject.emtree | Follow up | en_US |
dc.subject.emtree | Glioma | en_US |
dc.subject.emtree | Human | en_US |
dc.subject.emtree | Human tissue | en_US |
dc.subject.emtree | Meningioma | en_US |
dc.subject.emtree | Nuclear magnetic resonance imaging | en_US |
dc.subject.emtree | Overall survival | en_US |
dc.subject.emtree | Protein determination | en_US |
dc.subject.emtree | Protein expression | en_US |
dc.subject.emtree | Seizure | en_US |
dc.subject.emtree | Survival time | en_US |
dc.subject.emtree | Brain tumor | en_US |
dc.subject.emtree | Cancer staging | en_US |
dc.subject.emtree | Case control study | en_US |
dc.subject.emtree | Female | en_US |
dc.subject.emtree | Glioma | en_US |
dc.subject.emtree | Male | en_US |
dc.subject.emtree | Meningioma | en_US |
dc.subject.emtree | Metabolism | en_US |
dc.subject.emtree | Pathology | en_US |
dc.subject.emtree | Prognosis | en_US |
dc.subject.mesh | Adult | en_US |
dc.subject.mesh | Biomarkers, tumor | en_US |
dc.subject.mesh | Case-control studies | en_US |
dc.subject.mesh | Brain neoplasms | en_US |
dc.subject.mesh | Enzyme-linked immunosorbent assay | en_US |
dc.subject.mesh | Female | en_US |
dc.subject.mesh | Follow-up studies | en_US |
dc.subject.mesh | Glioma | en_US |
dc.subject.mesh | Humans | en_US |
dc.subject.mesh | Male | en_US |
dc.subject.mesh | Meningeal neoplasms | en_US |
dc.subject.mesh | Meningioma | en_US |
dc.subject.mesh | Neoplasm proteins | en_US |
dc.subject.mesh | Neoplasm staging | en_US |
dc.subject.mesh | Prognosis | en_US |
dc.subject.mesh | Proteoglycans | en_US |
dc.subject.scopus | Dermatan Sulfate Proteoglycan; Human ESM1 Protein; Proteoglycan | en_US |
dc.subject.wos | Biotechnology & applied microbiology | en_US |
dc.subject.wos | Genetics & heredity | en_US |
dc.subject.wos | Medicine, research & experimental | en_US |
dc.subject.wos | Pathology | en_US |
dc.title | Expressions of endocan in patients with meningiomas and gliomas | en_US |
dc.type | Article |