Publication:
Insulinoma case admitted with reactive hypoglycemia symptoms

dc.contributor.authorKoca, Nizameddin
dc.contributor.authorUgraş, Nesrin
dc.contributor.buuauthorCander, Soner
dc.contributor.buuauthorCANDER, SONER
dc.contributor.buuauthorGül, Ozen Oz
dc.contributor.buuauthorÖZ GÜL, ÖZEN
dc.contributor.departmentBursa Uludağ Üniversitesi/Tıp Fakültesi/Patoloji Anabilim Dalı.
dc.contributor.departmentBursa Uludağ Üniversitesi/Tıp Fakültesi/Endokrinoloji ve Metabolizma Anabilim Dalı.
dc.contributor.researcheridAAI-1005-2021
dc.date.accessioned2024-07-05T06:04:46Z
dc.date.available2024-07-05T06:04:46Z
dc.date.issued2020-01-01
dc.description.abstractAim: To present an insulinoma case with post-prandial hypoglycemic symptoms associated with glucose inducible endogenous hyperinsulinemia.Case: A 52-year-old female patient was evaluated for hypoglycemic symptoms especially those occuring within 3 hours after consuming sugary foods. These symptoms were persistent for a year and a half. She was diagnosed with reactive (post-prandial) syndrome and followed a recommended diet and was given acarbose but there was no improvement. The results suggested post-prandial endogenous hyperinsulinemia related hypoglycemia. Multiphasic computerized tomography revealed an 11x15x12 mm size mass lesion, anteriorly in the head and uncinate process of the pancreas and then the patients were treated surgically with pancreatic enucleation and cured.Conclusion: Distinguishing post-prandial syndrome by careful history and clinical evaluation in patients with postprandial symptoms is of great importance in terms of cost-effectivity. However, it should not be forgotten that although organic pathologies are mostly presented with fasting hypoglycemia, they may also cause post-prandial symptoms. Severity and progression of the symptoms that point to neuroglycopenia is important, and in this condition the most convenient clinical approach to the patient should be performed with careful and appropriate assessment steps.
dc.identifier.doi10.2174/1573399815666190712200716
dc.identifier.endpage796
dc.identifier.issn1573-3998
dc.identifier.issue7
dc.identifier.startpage790
dc.identifier.urihttps://doi.org/10.2174/1573399815666190712200716
dc.identifier.urihttps://hdl.handle.net/11452/42936
dc.identifier.volume16
dc.identifier.wos000569064900006
dc.indexed.wosWOS.ESCI
dc.language.isoen
dc.publisherBentham Science Publ
dc.relation.journalCurrent Diabetes Reviews
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi
dc.rightsinfo:eu-repo/semantics/closedAccess
dc.subjectArterial calcium stimulation
dc.subjectFunctioning insulinoma
dc.subjectManagement
dc.subjectPatient
dc.subjectNesidioblastosis
dc.subjectTerm
dc.subjectInsulinoma
dc.subjectReactive hypoglycemia
dc.subjectPost-prandial hypoglycemia
dc.subjectHyperinsulinemia
dc.subjectHypothyroidism
dc.subjectDiabetes
dc.subjectScience & technology
dc.subjectLife sciences & biomedicine
dc.subjectEndocrinology & metabolism
dc.titleInsulinoma case admitted with reactive hypoglycemia symptoms
dc.typeArticle
dspace.entity.typePublication
relation.isAuthorOfPublication7e655938-5300-4433-810e-24945b8c2774
relation.isAuthorOfPublication4ebb27f5-06de-45b8-8773-ea3452507df3
relation.isAuthorOfPublication.latestForDiscovery7e655938-5300-4433-810e-24945b8c2774

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